Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

We have reported that murine leukemia cell line (C2M-A5) induced apoptosis by G-CSF. To clarify the mechanism, mRNA expression of apoptosis-related genes was studied. It revealed transient over-expression of c-myc, H-ras and p53 and down-expression of bcl-2. These changes were known as triggers of endonuclease induction. After 96 h culture with G-CSF, apoptosis was occurred simultaneously with endonuclease (37 kd) activation. This endonuclease induced the digestion of double-strand DNA and might be associated with caspase3. Although G-CSF accelerates cell growth and prevents apoptosis in general, it is a contradictory effect. We concluded that G-CSF induced endogenous endonuclease activity in C2M-A5.

Type

Journal article

Journal

Leuk Res

Publication Date

12/2000

Volume

24

Pages

1033 - 1039

Keywords

Animals, Apoptosis, Caspase 3, Caspases, Endonucleases, Gene Expression, Genes, bcl-2, Genes, myc, Genes, p53, Genes, ras, Granulocyte Colony-Stimulating Factor, Leukemia, Myeloid, Mice, RNA, Messenger, Tumor Cells, Cultured